We have been taught, as have our physicians, that weight gain means diabetes. Let us look at the actual research, but first let us look at some basic facts about diabetes and obesity.
5 Important Facts About Diabetes
- Diabetes is not a disease – it is a symptom.
- Diabetes can, in fact, be reversed – it is not a lifelong, downhill process.
- The most effective medications, with all of their risks, are based on isolating compounds from herbs and synthesizing them (pharmaceutical industry can not make money on the real thing).
- Type 2 diabetes is not a life-sentence. New research has found that it is a symptom that can be reversed, and you can be free of it for the rest of your life if you keep your weight down.
- Type 2 diabetes actually has a variety of causes – which is why most obese people do not have diabetes!!
What Causes Diabetes?
There are several causes of the diabetic profile. Have you ever noticed how many people who are thin have diabetes! Diabetes can be caused by:
- Different types of infections or adrenal overload.
- Too much sugar in the diet or eating too many foods that quickly turn into sugar.
- High sugar intake (or the intake of AGEs) that creates AGEs in the body. AGEs are Advanced Glycation Endproducts caused when there is too much of sugar, and sugar starts combining with fats or proteins without an enzymatic process. These AGEs are far more dangerous than free radicals as they attach to cells, anywhere in the body, and cause dysfunction, cancer or death of the cell.
- Liver dysfunction or gut issues (i.e. imbalance or depletion of the microbiota).
- Several different types of hormonal imbalances.
Weight And Diabetes
Over 70% of obese people ‘Do Not’ have diabetes. The connection between weight and diabetes works in the following manner:
If your weight is predominantly due to sugar intake – you have a high probability of developing diabetes. When your body turns to utilizing just sugar for fuel; it stops using fats for fuel.
Anyone with a basic understanding of the fuel-making process in the cells understands that there are 5 different pathways that lead into the gluconeogenesis or glycolysis pathways, Krebs cycle and the ETC (Electron Transport Chain) that produces ATP (Adenosine Triphosphate).
When you stop using fat for fuel, you hold onto the fats. You need to retrain your body to use fats rather than sugars and voila and as a result, you will start losing the fat cells.
Professor Roy Taylor, Professor of Medicine and Metabolism at Newcastle University stated – “This supports our theory of a personal fat threshold. If a person gains more weight than they personally can tolerate, then diabetes is triggered, but if they then lose that amount of weight, then they go back to normal”.
Why Is This The Case?
It seems to depend on how much fat is stored in the pancreas. If you have a fatty pancreas, it prohibits the pancreas from making insulin – which of course is one of the many end issues causing a diabetic profile.
Long before the pancreas, we need to work with the gut, the liver, and the adrenals. Professor Taylor acknowledged that even long-standing diabetes can be eliminated.
The unfortunate part of the issue is that people who take Metformin (an anti-diabetic drug based on the synthetic compound found in many herbs) deplete their bodies of CoQ10 (just like the statin drugs do and most anti-depressants do) which causes havoc throughout the body because cells need CoQ10 to make fuel.
But the good part of the issue is that any herbalist, doctor of natural medicine, naturopath, etc, knows this basic stuff and what herbs to give in what dosage to reverse diabetes.
On the other hand, your diet usually has to work in combination with protocol although the diet may be very different depending on what is causing diabetes. For instance, the diet may have different components if the diabetes is the result of liver dysfunction versus adrenal dysfunction.
A simple answer to this is – find a good health practitioner who actually does their research and can identify the cause and solve the underlying problem.